Questões de Inglês - Grammar

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    Because it is locked away inside the skull, the brain is hard to study. Looking at it requires finicky machines which use magnetism or electricity or both to bypass the bone. There is just one tendril of brain tissue that can be seen from outside the body without any mucking about of this sort. That is the retina. Look into someone’s eyes and you are, in some small way, looking at their brain.

    This being so, a group of researchers decided to study the structure of the eye for signs of cognitive decline. Changes in the brain, they reasoned, might lead to changes in the nervous tissue connected to it. They focused on a part of the eye called the retinal nerve-fibre layer (RNFL). This is the lowest layer of the retina and serves to link the light-sensitive tissue above to the synapses which lead to the brain. The team’s results show that people with a thin RNFL are more likely to fail cognitive tests than those with a thick one. They are also more likely to suffer cognitive decline as they age.

(www.economist.com, 30.06.2018. Adaptado.)

TEXTO:

The latest innovation that’s changing the way
you break a sweat is likely to have even the most
internet-savvy millennials starry-eyed with workout
wonder.Today marks the release of the interactive
[5] gym, Mirror ($1,495, plus a $39 monthly subscription).
    At first glance, the device looks like a sleek, no
frills reflective surface you might hang up to checkout
your leggings-sports bra combo before heading off to
spin class. Switch the device on however, and you’ll
[10] come face-to-face with a trainer who can lead you
through a full class of cardio, strength, yoga, Pilates,
barre, boxing, and stretch—all of which you can tailor
by time and skill-level to your personal fitness goals.
Visual feedback is at the center of the smart decor’s
[15] design. Not only can you see yourself and the trainer
as you perform each move in front of the device, but in
the live classes, the trainer can see you and offer you
feedback, like “Make sure you can see your toes in
chair pose!”
[20]    If you can’t catch one of the 50 or more real-time
sweat sessions offered per week, each category of
class is also available on-demand for midnight HIIT
(High Intensity Interval Training) sessions or spur of
the moment afternoon pick-me-ups.

Disponível em: https://www.wellandgood.com/good-sweat/mirror-athome-workout-equipment/. Acesso em: 05 mar. 2008. Adaptado.

Text

TEXTO - Lung Cancer In Non Smoker

    A group of respiratory medicine and public health experts are calling for lung cancer in never-smokers to be given greater recognition. Writing in the Journal of the Royal Society of Medicine, they say that lung cancer in people who have never smoked is under recognised and presents a diagnostic challenge, particularly for GPs seeking to balance overinvestigation with early diagnosis and high quality care.
    It is estimated that around 6,000 people in the UK who have never smoked die of lung cancer every year, greater than the numbers of people who die of cervical cancer (900), lymphoma (5,200), leukaemia (4,500) and ovarian cancer (4,200).
    Major contributors to lung cancers in never-smokers include second-hand smoke, occupational carcinogen exposure and outdoor pollution. Globally, the use of solid fuels for indoor cooking and second-hand smoke exposure are important contributions to lung cancer in never-smokers and disproportionately affect women
    Lead author, Professor Paul Cosford, Director for Health Protection & Medical Director, Public Health England, said: "This paper demonstrates an estimated 6,000 people who have never smoked die each year from lung cancer in the UK. This makes it, by itself, the eighth most common cause of cancer related death in the UK.
    "For too long having lung cancer has only been thought of as a smoking related disease. This remains an important association but, as this this work shows, the scale of the challenge means there is a need to raise awareness with clinicians and policy makers of the other risk factors including indoor and outdoor air pollution.
    "This is one reason why PHE published its review of the evidence and recommended specific actions local authorities can take to improve their air quality. By delivering on the promise of a clean air generation we can reduce the number of lung cancers among those who have never smoked."
    Co-author Professor Mick Peake, clinical director of the Centre for Cancer Outcomes, University College London Hospitals Cancer Collaborative, said: "Despite advances in our understanding, most people who have never smoked do not believe they are at risk and often experience long delays in diagnosis, reducing their chances of receiving curative treatment."
    Prof Peake added: "The stigma of smoking has been the major factor behind the lack of interest in, knowledge of and research into lung cancer. Therefore, in many ways, neversmokers who develop lung cancer are, as a result, disadvantaged.
    "Drawing attention to the contribution of underlying risk factors to lung cancer in never-smokers presents opportunities to reinforce efforts to tackle other major public health challenges. For example, the impact of passive smoking and air pollution on lung cancers adds weight to the government's ambitions to improve air quality and the public, clinicians and policy makers must all be aware of this relationship

(Adapted from: www.sciencedaily.com)

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The drugs don’t work: what happens after antibiotics?

Antibiotic resistance is growing so fast that routine surgery could soon become impossible. But scientists are fighting back in the battle against infection

1- The first antibiotic that didn’t work for Debbi Forsythe was trimethoprim. In March 2016, Forsythe, a genial primary care counsellor from Morpeth, Northumberland, contracted a urinary tract infection. UTIs are common: more than 150 million people worldwide contract one every year. So when Forsythe saw her GP, they prescribed the usual treatment: a three-day course of antibiotics. When, a few weeks later, she fainted and started passing blood, she saw her GP again, who again prescribed trimethoprim.

2- Three days after that, Forsythe’s husband Pete came home to find his wife lying on the sofa, shaking, unable to call for help. He rushed her to A&E. She was put on a second antibiotic, gentamicin, and treated for sepsis, a complication of the infection that can be fatal if not treated quickly. The gentamicin didn’t work either. Doctors sent Forsythe’s blood for testing, but such tests can take days: bacteria must be grown in cultures, then tested against multiple antibiotics to find a suitable treatment. Five days after she was admitted to hospital, Forsythe was diagnosed with an infection of multi-drug-resistant E coli, and given ertapenem, one of the so-called “last resort” antibiotics.

3- It worked. But damage from Forsythe’s episode has lingered and she lives in constant fear of an infection reoccurring. Six months after her collapse, she developed another UTI, resulting, again, in a hospital stay. “I’ve had to accept that I will no longer get back to where I was,” she says. “My daughter and son said they felt like they lost their mum, because I wasn’t who I used to be.” But Forsythe was fortunate. Sepsis currently kills more people in the UK than lung cancer, and the number is growing, as more of us develop infections immune to antibiotics.

4- Antimicrobial resistance (AMR) – the process of bacteria (and yeasts and viruses) evolving defense mechanisms against the drugs we use to treat them – is progressing so quickly that the UN has called it a “global health emergency”. At least 2 million Americans contract drug-resistant infections every year. So-called “superbugs” spread rapidly, in part because some bacteria are able to borrow resistance genes from neighbouring species via a process called horizontal gene transfer. In 2013, researchers in China discovered E coli containing mcr-1, a gene resistant to colistin, a last-line antibiotic that, until recently, was considered too toxic for human use. Colistin-resistant infections have now been detected in at least 30 countries.

5- “In India and Pakistan, Bangladesh, China, and countries in South America, the resistance problem is already endemic,” says Colin Garner, CEO of Antibiotic Research UK. In May 2016, the UK government’s Review on Antimicrobial Resistance forecast that by 2050 antibiotic-resistant infections could kill 10 million people per year – more than all cancers combined.

6- “We have a good chance of getting to a point where for a lot of people there are no [effective] antibiotics,” Daniel Berman, leader of the Global Health team at Nesta, told me. The threat is difficult to imagine. A world without antibiotics means returning to a time without organ transplants, without hip replacements, without many now-routine surgeries. It would mean millions more women dying in childbirth; make many cancer treatments, including chemotherapy, impossible; and make even the smallest wound potentially lifethreatening. As Berman told me: “Those of us who are following this closely are actually quite scared.”

7- Bacteria are everywhere: in our bodies, in the air, in the soil, coating every surface in their sextillions. Many bacteria produce antibiotic compounds – exactly how many, we don’t know – probably as weapons in a microscopic battle for resources between different strains of bacteria that has been going on for billions of years. Because bacteria reproduce so quickly, they are able to evolve with astonishing speed. Introduce bacteria to a sufficiently weak concentration of an antibiotic and resistance can emerge within days. Penicillin resistance was first documented in 1940, a year before its first use in humans. (A common misconception is that people can become antibioticresistant. They don’t – the bacteria do.)

Oliver Franklin-Wallis Sun 24 Mar 2019 In: https://www.theguardian.com/global/2019/mar/24/ the-drugs-dont-work-what-happens-after-antibiotics